Description
SARS-CoV-2’s spike protein begins its journey in the upper respiratory tract, where the virus initially takes hold. Once the mucosal barrier in the nose and throat is breached, the spike protein can enter the bloodstream and circulate throughout the body. From there, it spreads to major organs, including the brain, kidneys, liver, and intestines. Even after the virus is no longer active, the spike protein may linger in these tissues, potentially triggering silent inflammation and long-term damage. This spread is a significant concern, as it could explain persistent symptoms, such as fatigue and brain fog, reported by many individuals following COVID-19.
Research has found that the spike protein can settle in the skull and areas surrounding the brain, contributing to neuroinflammation and other neurological problems. In mouse models, the presence of spike protein alone was enough to induce brain changes and behavioral issues. Similarly, in humans, it has been linked to inflammation and markers of neurodegeneration, which may increase vulnerability to other brain injuries. These findings highlight the need to better understand the spike protein’s persistence and its long-term effects on both physical and mental health.
